Amoebas at the start
Were not complex;
They tore themselves apart
And started Sex.
Weekly cell and molecular biology metablogging:
The Discovery of the Neuron
by Mo at Neurophilosophy
by Ian at Mystery Rays From Outer Space
Faulty Cell Membrane Repair Causes Heart Disease
by Snowcrash at Biosingularity
How Cells Can Cheat Death
by Charles at Science and Reason
Yet Another Way that miRNAs Turn Off Transcripts
by Alex at The Daily Transcript
Unraveling How DNA Replicates
by Krishna on the Cornell Chronicle Online
IVF Strategies: Cloned Sperm and Preimplantation Genetic Diagnosis
by Hsien-Hsien at Eye on DNA
by dr.ina at Sporula
And some ScienceDaily picks, below the fold:
Scientists Identify A Mouse Embryonic Stem Cell More Like Our Own
Scientists have discovered a new type of mouse embryonic stem cell that is the closest counterpart yet to human embryonic stem (ES) cells, the National Institutes of Health (NIH) announced today. The cells are expected to serve as an improved model for human ES cells in studies of regeneration, disease pathology and basic stem cell biology.
One of the fundamental traits of a tumor — how it avoids the immune system — might become its greatest vulnerability, according to researchers from the University of Southern California. Their findings, demonstrated in human breast and colorectal cancers, indicate that a technique for determining a tumor’s “immune signature,” could be useful for diagnosing and treating specific cancers.
Fibrinogen, a blood-clotting protein found in circulating blood, has been found to inhibit the growth of central nervous system neuronal cells, a process that is necessary for the regeneration of the spinal cord after traumatic injury. The findings may explain why the human body is unable to repair itself after most spinal cord injuries.
During vigorous exercise, heart muscle cells take a beating. In fact, some of those cells rupture and without an efficient repair process those cells would die and cause heart damage (cardiomyopathy). University of Iowa researchers have discovered a specific repair mechanism in heart muscle and identified a protein called dysferlin that is critical for resealing heart muscle cell membranes.