The quote of the week is from Mark Twain. It is a bit off-topic, but it’s worth saying:
An inglorious peace is better than a dishonorable war.
Now, welcome to your weekly dose of cell and molecular biology. As always, I’ve selected all of the blogging commentary that I’ve seen, trying to keep the selection both topical and not mere reposting of press releases from jouranls and societies. The result, hopefully, is a zeitgeist of this week’s cyto-blogging:
Small Things Considered
Fungal Genomes And Comparative Genomics
- Jet lag: it’s all about chemical reactions in cells, Cornell researchers say in explaining biology of circadian clocks
And five picks from ScienceDaily below the fold:
Lab Defines Proteins That Distinguish Chromosome Ends From DNA Double-strand Breaks:
Scientists offer insight into the way cells protect chromosome ends from misguided repair.
Who must go? When to go? Where to go? During development ovarian cells migrate in a spacial-temporal coordinated way, responding to specific signals that determine which cells have to move, when they have to move, and where they have to go.
Nerve cells grown in three-dimensional environments deploy hundreds of different genes compared with cells grown in standard two-dimensional petri dishes, according to a new Brown University study. The research adds to a growing body of evidence that lab culture techniques dramatically affect the way these cells behave.
Researchers have constructed the first global family tree of metabolic protein architecture. Their approach offers a new window on the evolutionary history of metabolism.
Scientists have identified the cells that cause Ewing’s sarcoma. They are cells of the mesenchyme, the connective tissue that supports other tissues. The researchers have also succeeded in transforming the tumor cells into virtually normal mesenchymal cells again. These results open up new therapeutic possibilities for blocking the development of Ewing’s sarcoma in young patients.